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Phagocytes and infection

Mikael Rhen

Phagocyte movement is central facet in our immune defense. However, selected bacterial intracellular infections hijack phagocytes for systemic spread. As many bacterial virulence factors specifically interfere with actin polymerization, one could envision that such virulence factors also affect phagocyte movement. Nanoscale bio-electronic cell culture devices open exciting possibilities to dissect connections between the ability of selected model pathogens - such as Burholderia thailandensis, Mycobacterium bovis and Salmonella enterica - and their virulence factors to interfere with phagocyte motility and chemotactic responses. The development of such systems will be applicable for a number of research fields involved in phagocyte biology and cellular microbiology.

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